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SELECT showed that group gets significant MACE reduction, which is a mortality-relevant outcome

Hyperaldosteronism is associated with impaired vascular reactivity, and it has been shown that aldosterone induces a G6PD-deficient phenotype to impair endothelial function, and aldosterone antagonism or gene transfer of G6PD improves vascular reactivity [9]

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doi: 10.1200/jco.2008.16.3725 24 RochlinDHBarrioAVMcLaughlinSZee VanKJWoodsJFDayanJHet al

Further, Folkman isolated a tumor-derived factor that induced angiogenesis [2] and hypothesized that inhibition of angiogenic signaling pathways might block new vessel formation and result in tumor dormancy