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Botham, PhD, DSc & Peter A

From a molecular point of view, the necroptosis pathway is initiated by auto- and trans-phosphorylation of RIPK1 and RIPK3 at Ser-227, resulting in stimulation of their kinase activity and assembly of a heterodimer protein complex termed necrosome via their RIPK homotypic interaction motif domains (Oliveira et al., 2018

doi: 10.1186/s40364-024-00593-x 19 ChenL.MinJ.WangF

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496 Conditional and neurone specific deletion of GDI1 (CamkII-Cre + -Gdi1 flox/Y model), showed that the down regulation of GDI in neurones in adult forebrain regions was sufficient to recapitulate the learning deficits previously shown in Gdi1 -null mice